Question: (Hi Doctor--I attempted to respond directly, but it seems that the website has maxed out on that particular conversation thread. I will send you my reply as a separate new question here.)

* * * * * * *

"Dr Vaishalee,

Your capacity to behold the elements of this case in a clear and knowing manner is one of the most unique things I have experienced in an MD. You are brilliant.

Yes--the crystallization by particle attachment (and thus growth) theory, as applied to pharmaceuticals or drugs saturated within the organism, is indeed a plausible theory. I establish more support for this theory in my original compiled chronicles--the C-set. Included in that support are references to other cases where people are, in fact, visibly growing various solidified supersaturates out from their eyes, as well as cases of drug flashbacks and return of symptoms that, likely, may be more than just need flashbacks.

. . .

You are correct in its ability to saturate the intracellular spaces: Phenibut F.A.A. is a synthetic aromatic amino acid, and as such is highly organic and fat-soluble. Phenibut alone, however, does not seem to produce these symptoms is any other cases that I have researched or heard about; thus I can only assume that it is the supplemental, negatively-synergisyic of Fasoracetam as a hyper-sensitizer of GABA-B, plus the strong possibility of crystal growth of saturated within the ion-rich environment of the body, that fuels the continuation of drug presence and effects.

You are correct-- the lack of immunity and inflammation does seem to play a role in perpetuating an effective masking of the underlying conditions. Not only with this drug toxicity, but with further damage caused over a year ago, by excessive consumption of cocaine. More on this in the original C-set documents.

A trial of extracorporeal detoxification would be a miracle! In my own proposals for a comprehensive treatment plan, contained in the C-set, I include dialysis (along with albumin-based treatments and hemoperfusion) to cause for the most effective and broad-spectrum removal of likely-harmful solutes in the body. Dialysis and hemoperfusion are both part of the latest comprehensive detox systems done in the West, called M.A.R.S. I go into a deeper analysis of the potential of each form of treatment in my writings. (In fact, such removal may be most indicated, as a lack of metabolization has recently resulted in apparent pooling of toxins in flesh so strong, that several spots of burnt hair and damaged nerve endings on skin have spontaneously formed, in addition to pH of urine that is often too extremely alkaline. More on the burnt missing hair patches and numb spots in the documents.)

Fasting is an effective form of releasing these agents, and allowing them to enter the circulation from deep tissue storage. Also effective, as you correctly assess, is fat immersion.

I was stuck with a complete lack of withdrawals for over one year. Do you know when I experienced my first wave of spontaneous withdrawal? It was after doing "olive oil" flush, consumed orally. Additionally, since then, even oral oil pulling with coconut oil has produced strong (although temporary) withdrawals, as desaturating the gums and mouth (places numbed by coke for almost 2 years without having taken it) provides a better ability for remaining solutes to diffuse and lower their concentration slightly within the surrounding tissue volumes.

You are absolutely correct about opportunistic infections. I have had multiple issues with infections, although most have remained undiagnosed. Undiagnosed selective IgM deficiency seems to be the result of the GABAergic overwhelm and CNS suppression, as IgM has been found to be below standard range in the presence of multiple infections, while other markers also remained unelevated.

Doctor, you are absolutely correct about using distilled water with lowest T.D.S. It has been my experience that such water is several times more effective at pulling solutes from tissues and inducing temporary withdrawals, as regular spring or tap water.

. . .

Your intellect and strong, accurate intuition are a tremendous source of hope and comfort to me. You cannot grasp how grateful I feel for your wisdom and clear understanding of the matters.

I trust you, with the most private material I have compiled on the matter. It is a large request to ask, as the number of pages within the C-set total up to a few hundred. It is the most complete written work, summary and analysis that I have prepared on the matter.

For such a thorough reading into this final, last remaining set of information, this may place you in the position of becoming the world's leading MD expert on the subject matters pertaining to and intersecting at this one parricular case of mine.

For such an examination of my written work, which may reasonably take one at least a few days to read through at a reasonable pace, I would be willing to provide additional compensation or gift to you, happily, in gratitude for the generous gift of not only your professional but personal interest and time.

I truly believe that after assessing this final set, we may find ourselves be holding a case with such merit and unique position, that it should warrant even widespread, worldwide medical attention (even social media, etc)--as well as challenge old theories and help to shape new ones within the system of medicine.

. . .

I shall sent you the compressed file for the C-set very shortly.

To preface the material, and to help establish the proper context, I will mention a few key things.

(1) These are chronicled documents composed over time, starting back in 2016, and most recently being worked on now for an update between July thru the present time.

(2) In the earlier parts and documents, I didnt yet learn about hepatic encephalopathy, or most other things. Thus, in reading my experiences, both my writing style and approach to the issues reveal a relative immaturity and slight lack of confidence in what I present, which other readers have picked up on and told me as feedback.

(3) By around the middle documents of the set, my level of understanding and confidence in writing become noticeably stronger, and feedback from others reveals that it is in these middle to later stages and documents that everything is wrapped up much more strongly. (It is for that reason of initially-weak presentation that I found a separate "B-set" summary to be necessary, as it does a better job of summarizing and presenting the issues and theories than the earlier parts of the C-set, which remain so far unedited.)

(4) A thorough reading, from beginning to end and in order, will provide the overall best level of understanding; this understanding does not come at the strongest levels until the latter documents are read. Additionally, however, the latter documents make no sense unless the earlier documents are read first, through which only a healthy "suspension of disbelief" and an open mind may inoculate the reader against a premature and unwarranted doubtfulness. (Hopefully, after reading the A-set and B-set, the reader already has an open mind.)

(5) The two testimonials produced by two other women, have both been produced after each of them were able to read through the entirety of the C-set. Their conclusions, resultingly, came to the highest level of certainty, as they both presented, only after reading the deeper details within the C-set, while acting as kinds of judges or investigators. Their favorable opinion is a testament to the strength of what is presented in the C-set, when read in its entirety.

(6) To restate: the earlier documents may be slightly more difficult to process and read thru, and also contain older, and in select cases outdated narratives and theories. However, once reaching the middle and latter documents, the reader should start seeing many things clicking together. As one person told me, "The first read-through, we aren't that sure at first what to make of this in the beginning. It is like a good mystery novel or movie, where you watch the first 90 percent and have no idea what is really going on. Then, further in, it finally "clicks", with a big A-HA moment, like the ending of a movie that reveals the truth clearly. Afterwards, when I go to re-read this C-set again a second time, it is like a completely different experience, because this time I can see how every single thing you said all along fit perfectly with what we now know. It is truly remarkable phenomenon to experience this, which can only be done with both patience and faith that there is more unifying brilliance to the material than what one may first see."

. . .

Doctor, today I deal with more than just Phenibut/Fasoracetam poisoning and low respiratory rate. As a result of various complications, I deal with acute on chronic apparent intestinal perforation, onset of ankylosing spondylitis, among others; all are undiagnosed. A closer examination of the facts presented may yield a better insight into the nature and causes of these.

Doctor, you are the only licensed professional that I have encountered, who has taken an interest in this case with a positive, favorable analysis. I am very excited to see what you think after carefully examining this last set of material that I have produced on this subject matter.

Thank you for everything."

(Hi Doctor--I attempted to respond directly, but it seems that the website has maxed out on that particular conversation thread. I will send you my reply as a separate new question here.)

* * * * * * *

"Dr Vaishalee,

Your capacity to behold the elements of this case in a clear and knowing manner is one of the most unique things I have experienced in an MD. You are brilliant.

Yes--the crystallization by particle attachment (and thus growth) theory, as applied to pharmaceuticals or drugs saturated within the organism, is indeed a plausible theory. I establish more support for this theory in my original compiled chronicles--the C-set. Included in that support are references to other cases where people are, in fact, visibly growing various solidified supersaturates out from their eyes, as well as cases of drug flashbacks and return of symptoms that, likely, may be more than just need flashbacks.

. . .

You are correct in its ability to saturate the intracellular spaces: Phenibut F.A.A. is a synthetic aromatic amino acid, and as such is highly organic and fat-soluble. Phenibut alone, however, does not seem to produce these symptoms is any other cases that I have researched or heard about; thus I can only assume that it is the supplemental, negatively-synergisyic of Fasoracetam as a hyper-sensitizer of GABA-B, plus the strong possibility of crystal growth of saturated within the ion-rich environment of the body, that fuels the continuation of drug presence and effects.

You are correct-- the lack of immunity and inflammation does seem to play a role in perpetuating an effective masking of the underlying conditions. Not only with this drug toxicity, but with further damage caused over a year ago, by excessive consumption of cocaine. More on this in the original C-set documents.

A trial of extracorporeal detoxification would be a miracle! In my own proposals for a comprehensive treatment plan, contained in the C-set, I include dialysis (along with albumin-based treatments and hemoperfusion) to cause for the most effective and broad-spectrum removal of likely-harmful solutes in the body. Dialysis and hemoperfusion are both part of the latest comprehensive detox systems done in the West, called M.A.R.S. I go into a deeper analysis of the potential of each form of treatment in my writings. (In fact, such removal may be most indicated, as a lack of metabolization has recently resulted in apparent pooling of toxins in flesh so strong, that several spots of burnt hair and damaged nerve endings on skin have spontaneously formed, in addition to pH of urine that is often too extremely alkaline. More on the burnt missing hair patches and numb spots in the documents.)

Fasting is an effective form of releasing these agents, and allowing them to enter the circulation from deep tissue storage. Also effective, as you correctly assess, is fat immersion.

I was stuck with a complete lack of withdrawals for over one year. Do you know when I experienced my first wave of spontaneous withdrawal? It was after doing "olive oil" flush, consumed orally. Additionally, since then, even oral oil pulling with coconut oil has produced strong (although temporary) withdrawals, as desaturating the gums and mouth (places numbed by coke for almost 2 years without having taken it) provides a better ability for remaining solutes to diffuse and lower their concentration slightly within the surrounding tissue volumes.

You are absolutely correct about opportunistic infections. I have had multiple issues with infections, although most have remained undiagnosed. Undiagnosed selective IgM deficiency seems to be the result of the GABAergic overwhelm and CNS suppression, as IgM has been found to be below standard range in the presence of multiple infections, while other markers also remained unelevated.

Doctor, you are absolutely correct about using distilled water with lowest T.D.S. It has been my experience that such water is several times more effective at pulling solutes from tissues and inducing temporary withdrawals, as regular spring or tap water.

. . .

Your intellect and strong, accurate intuition are a tremendous source of hope and comfort to me. You cannot grasp how grateful I feel for your wisdom and clear understanding of the matters.

I trust you, with the most private material I have compiled on the matter. It is a large request to ask, as the number of pages within the C-set total up to a few hundred. It is the most complete written work, summary and analysis that I have prepared on the matter.

For such a thorough reading into this final, last remaining set of information, this may place you in the position of becoming the world's leading MD expert on the subject matters pertaining to and intersecting at this one parricular case of mine.

For such an examination of my written work, which may reasonably take one at least a few days to read through at a reasonable pace, I would be willing to provide additional compensation or gift to you, happily, in gratitude for the generous gift of not only your professional but personal interest and time.

I truly believe that after assessing this final set, we may find ourselves be holding a case with such merit and unique position, that it should warrant even widespread, worldwide medical attention (even social media, etc)--as well as challenge old theories and help to shape new ones within the system of medicine.

. . .

I shall sent you the compressed file for the C-set very shortly.

To preface the material, and to help establish the proper context, I will mention a few key things.

(1) These are chronicled documents composed over time, starting back in 2016, and most recently being worked on now for an update between July thru the present time.

(2) In the earlier parts and documents, I didnt yet learn about hepatic encephalopathy, or most other things. Thus, in reading my experiences, both my writing style and approach to the issues reveal a relative immaturity and slight lack of confidence in what I present, which other readers have picked up on and told me as feedback.

(3) By around the middle documents of the set, my level of understanding and confidence in writing become noticeably stronger, and feedback from others reveals that it is in these middle to later stages and documents that everything is wrapped up much more strongly. (It is for that reason of initially-weak presentation that I found a separate "B-set" summary to be necessary, as it does a better job of summarizing and presenting the issues and theories than the earlier parts of the C-set, which remain so far unedited.)

(4) A thorough reading, from beginning to end and in order, will provide the overall best level of understanding; this understanding does not come at the strongest levels until the latter documents are read. Additionally, however, the latter documents make no sense unless the earlier documents are read first, through which only a healthy "suspension of disbelief" and an open mind may inoculate the reader against a premature and unwarranted doubtfulness. (Hopefully, after reading the A-set and B-set, the reader already has an open mind.)

(5) The two testimonials produced by two other women, have both been produced after each of them were able to read through the entirety of the C-set. Their conclusions, resultingly, came to the highest level of certainty, as they both presented, only after reading the deeper details within the C-set, while acting as kinds of judges or investigators. Their favorable opinion is a testament to the strength of what is presented in the C-set, when read in its entirety.

(6) To restate: the earlier documents may be slightly more difficult to process and read thru, and also contain older, and in select cases outdated narratives and theories. However, once reaching the middle and latter documents, the reader should start seeing many things clicking together. As one person told me, "The first read-through, we aren't that sure at first what to make of this in the beginning. It is like a good mystery novel or movie, where you watch the first 90 percent and have no idea what is really going on. Then, further in, it finally "clicks", with a big A-HA moment, like the ending of a movie that reveals the truth clearly. Afterwards, when I go to re-read this C-set again a second time, it is like a completely different experience, because this time I can see how every single thing you said all along fit perfectly with what we now know. It is truly remarkable phenomenon to experience this, which can only be done with both patience and faith that there is more unifying brilliance to the material than what one may first see."

. . .

Doctor, today I deal with more than just Phenibut/Fasoracetam poisoning and low respiratory rate. As a result of various complications, I deal with acute on chronic apparent intestinal perforation, onset of ankylosing spondylitis, among others; all are undiagnosed. A closer examination of the facts presented may yield a better insight into the nature and causes of these.

Doctor, you are the only licensed professional that I have encountered, who has taken an interest in this case with a positive, favorable analysis. I am very excited to see what you think after carefully examining this last set of material that I have produced on this subject matter.

Thank you for everything."

Hi Doctor,

I will respond within about 48 hours to each round to keep the thread alive, and also allow for reading without much rush.

I will tell you something. For a long gimr, my family (primarily my mother) thought I was crazy, and just making up symptoms, or suffering from something purely "psychological" or psychiatric. Despite the testimonies, she did not think much differently.

Two things have helped recently change that. Firstly, the set of 4 documents I wrote on ammonia and HE; and, secondly, your very own endorsement of my case, and of a proper diagnosis.

Now, my own mother is admitting that I may have been correct all along that I have been dealing with legitimate medical issues. As a result, she has also started to read carefully through the same C-set documents, which she is reading this week! She is currently up to about C10 or C11.

As for Ankylosing spondylitis, a much more deep explanation is included in the most recent documents (as it had onset only recently). Yes, I have been doing yoga regularly for years. Unfortunately, the issue causes the range of motion to become more restricted at the discs and joints. It has restricted my body very much. Time will tell what happens.

Doctor, you are right. Recreational drugs are not the best idea for me, especially given the position I am in. I am proud to state that I have been sober (ie, not having dosed anything) in over a year!

As for the intestinal complications... I wish I could confirm that it has only been flora issues at the root of it. Unfortunately, what is both the most certain and apparent to me, and the LEAST apparent or likely by anyone else, is the fact that I've been living with an "acute abdomen" for over a year.

It is an unbelievable thing to behold. But I assure you it is 100 percent true. Friends have asked me how certain I am of this; I have made it clear, that of all the issues I've dealt with (Phenibut poisoning, etc), the one thing that I am the MOST sure of, literally 100.000% with virtually no margin for error, is that I've experienced and lived with intestinal and peritoneal tear.

The details and accounts in the writings will make this clear, over time, as all the facts spread over the several or more documents are all assessed together.

It is the most dangerous thing I currently deal with--not the original substance toxicities themselves...!

I have always strived towards alkalinity earlier in my life, as we are always told that it is healthier than approaching the state of excessive acidity. Yet, ironically enough, and perhaps not what one would expect in my case, is that consistently over the last couple of years, my urine has been alkaline, often at almost 9.0 pH. This may be too basic! The literature, too, points out that alkalininity in the body causes drugs to be retained for much longer-- and that acidification helps increase the excretion rates of various compounds.

I've had multiple experiences that have supported this, and I have found this to be true. Perhaps in a safe supervised setting, acidification tactics may be used alongside extracorporeal methods (or even sweating) to increase drug and toxin excretion.

Oil massages are a fantastic idea! I have harbored ideas of this for some time, as the oil may pull many of the solutes out. Even warm water baths do the same, as they cause strong withdrawals.

Soft fruits and juices pretty much consist of the entirety of my diet today, as I have been very cautious ever since the cocaine-induced intestinal tears have taken place.

Thank you for your dedication. Please do not feel the need to rush, as slower, more careful and deliberate reading may be best. Looking forward to hearing more from you within a day or two.

Many blessings to you, Doctor.

Hi Doctor,

I will respond within about 48 hours to each round to keep the thread alive, and also allow for reading without much rush.

I will tell you something. For a long gimr, my family (primarily my mother) thought I was crazy, and just making up symptoms, or suffering from something purely "psychological" or psychiatric. Despite the testimonies, she did not think much differently.

Two things have helped recently change that. Firstly, the set of 4 documents I wrote on ammonia and HE; and, secondly, your very own endorsement of my case, and of a proper diagnosis.

Now, my own mother is admitting that I may have been correct all along that I have been dealing with legitimate medical issues. As a result, she has also started to read carefully through the same C-set documents, which she is reading this week! She is currently up to about C10 or C11.

As for Ankylosing spondylitis, a much more deep explanation is included in the most recent documents (as it had onset only recently). Yes, I have been doing yoga regularly for years. Unfortunately, the issue causes the range of motion to become more restricted at the discs and joints. It has restricted my body very much. Time will tell what happens.

Doctor, you are right. Recreational drugs are not the best idea for me, especially given the position I am in. I am proud to state that I have been sober (ie, not having dosed anything) in over a year!

As for the intestinal complications... I wish I could confirm that it has only been flora issues at the root of it. Unfortunately, what is both the most certain and apparent to me, and the LEAST apparent or likely by anyone else, is the fact that I've been living with an "acute abdomen" for over a year.

It is an unbelievable thing to behold. But I assure you it is 100 percent true. Friends have asked me how certain I am of this; I have made it clear, that of all the issues I've dealt with (Phenibut poisoning, etc), the one thing that I am the MOST sure of, literally 100.000% with virtually no margin for error, is that I've experienced and lived with intestinal and peritoneal tear.

The details and accounts in the writings will make this clear, over time, as all the facts spread over the several or more documents are all assessed together.

It is the most dangerous thing I currently deal with--not the original substance toxicities themselves...!

I have always strived towards alkalinity earlier in my life, as we are always told that it is healthier than approaching the state of excessive acidity. Yet, ironically enough, and perhaps not what one would expect in my case, is that consistently over the last couple of years, my urine has been alkaline, often at almost 9.0 pH. This may be too basic! The literature, too, points out that alkalininity in the body causes drugs to be retained for much longer-- and that acidification helps increase the excretion rates of various compounds.

I've had multiple experiences that have supported this, and I have found this to be true. Perhaps in a safe supervised setting, acidification tactics may be used alongside extracorporeal methods (or even sweating) to increase drug and toxin excretion.

Oil massages are a fantastic idea! I have harbored ideas of this for some time, as the oil may pull many of the solutes out. Even warm water baths do the same, as they cause strong withdrawals.

Soft fruits and juices pretty much consist of the entirety of my diet today, as I have been very cautious ever since the cocaine-induced intestinal tears have taken place.

Thank you for your dedication. Please do not feel the need to rush, as slower, more careful and deliberate reading may be best. Looking forward to hearing more from you within a day or two.

Many blessings to you, Doctor.

Hi Doctor,

Indeed, one of the very few things I never became fully certain around, was what this white material ended up being. I have not experienced or reported this in the recent times. I may have assumed it could have been a multiple number of things, and left the matter untouched.

You mentioned in a previous message the aparent phantom-nature of the smells I experience. This is a common misperception among first-time readers, as I may not have made sure to be clear enough in the beginning documents about what it is.

Eventually, I have made clear to family, friends and others readers, this clear and certain fact: my enhanced sense of smell causes me to perceive scents at lower thresholds that are not phantom, but completely real.

This is a result of the increased GABAergic tone, as neurologists have found that increased GABA tone (particularly GABA-B) increases the accuracy and power of the sense of smell.

You know how the bottoms of plastic containers have triangles with numbers, like 1, 2, 4, 5, 7 etc? My sense of smell, when on strong drug release, is so high, that I am able to differentiate between the different types of plastics by smell I have demonstrated this to friends, smelling and correctly differentiating types of plastic--some of which I can smell from dozens of meters away, in correct conditions.

I can also smell the types of medications people take through their skin and breath, if I am already familiar with the odor of that medication. Multiple times, people were shocked when I told them I knew the name of the medication they were on and then told them--they almost started to cry at how I could know such a private thing. Drugs are the same--I can smell who is carrying illicit drugs in public.

This is the magnitude to which my GABA system has been upregulated. Vision has had similar enhancements--I could see nearly everything out of my peripheral vision. However, one thing for sure, is that there has been no experience or evidence of any "false scents" or phantom smells, or of any smells that do not have a real, specific and true origin in reality, from a specific source around me. Pretty wild, huh?

To further elaborate, those phantom smells, associated with delusions and schizophrenia, are the symptom of a class of psychotic and schizophrenic-like conditions which are associated almost universally with a neurological imbalance characterized by LOW GABA tone (this is why people become temporary schizophrenic-like delusionals during alcohol or benzos withdrawals); however, with high GABA tone, which is the exact opposite issue which is the one that I have, this acts as a type of condition that nootropic drugs bring on, causing enhanced, accurate perception of reality.

As for the acute abdomen, doctors have denied it and not taken the issues seriously, for the reason that no one GI doctor or surgeon has bothered to fully understand the nature of the case. They just think, "symptom, diagnosis. Symptom, diagnosis," and cannot escape or expand from this one-dimensional thinking to see the bigger picture. But my life and well-being are on the line.

Yes, I've accepted that much damage may not be reversed. I just have to live with what I have, and get the proper medical attention.

I have considered either enteroscopy or a stand-up MRI using pineapple juice as an oral contrast (very high signal on T2-weighted images due to manganese content, one can find the studies and articles on this for more info), to show if the juice flows out of the places it is supposed to be in, and into places that it isn't supposed to end up in.

Adding ghee, or any oil or fatty liquid substance, has both a positive and negative. I have considered the benefits offered by withdrawals caused by the introduction of the fats. However, for most types of lipids, proper digestion and assimilation can only occur within the digestive tract, through bile and other active liver and digestive action to conjugate the oils. For this reason, most oils may cause problems when They are allowed to escape the GI tract, in the event that they end up in extra-intestinal space, as the body may not be effective at re-uptaking and processing these oils efficiently.

However, according to my best research, the types of oils that bypass the digestive requirement for most lipjds, are MCTs, or medium-chain triglycerides (specifically C8 and C10, or capric and acrylic oils, both derived from coconut oils). These oils are of such a structure that they can diffuse through and across cells and membranes and assimilate directly into the body without the need for digestion, thus people with digestive deficiencies are able to utilize this sub-class of oils.

I have used coconut oil products and purified, refined C8/C10 oils. They also induce withdrawals.

But, eventually, symptoms inevitably return. To date, three years later, my best two theories remain: either Fasoracetam alone continues to perpetually upregulate receptor density-- and thus the perception--of increased drug effects--or, in fact, it really is true (as I believe and have accumulated more evidence for) that crystal growth is taking place, following the initial nucleations and saturation from 2016.

Doctor, your mind on this matter, I find, may be invaluable to me.

At the conclusion of your reading of this, I would like to offer to hire you, for a reasonable stipend paid regularly, to be a continued personal medical consultant to me. My friends and family are literally celebrating the fact that the first "MD" takes my case seriously. I expect that we can open many doors together.

-Jon

Hi Doctor,

Indeed, one of the very few things I never became fully certain around, was what this white material ended up being. I have not experienced or reported this in the recent times. I may have assumed it could have been a multiple number of things, and left the matter untouched.

You mentioned in a previous message the aparent phantom-nature of the smells I experience. This is a common misperception among first-time readers, as I may not have made sure to be clear enough in the beginning documents about what it is.

Eventually, I have made clear to family, friends and others readers, this clear and certain fact: my enhanced sense of smell causes me to perceive scents at lower thresholds that are not phantom, but completely real.

This is a result of the increased GABAergic tone, as neurologists have found that increased GABA tone (particularly GABA-B) increases the accuracy and power of the sense of smell.

You know how the bottoms of plastic containers have triangles with numbers, like 1, 2, 4, 5, 7 etc? My sense of smell, when on strong drug release, is so high, that I am able to differentiate between the different types of plastics by smell I have demonstrated this to friends, smelling and correctly differentiating types of plastic--some of which I can smell from dozens of meters away, in correct conditions.

I can also smell the types of medications people take through their skin and breath, if I am already familiar with the odor of that medication. Multiple times, people were shocked when I told them I knew the name of the medication they were on and then told them--they almost started to cry at how I could know such a private thing. Drugs are the same--I can smell who is carrying illicit drugs in public.

This is the magnitude to which my GABA system has been upregulated. Vision has had similar enhancements--I could see nearly everything out of my peripheral vision. However, one thing for sure, is that there has been no experience or evidence of any "false scents" or phantom smells, or of any smells that do not have a real, specific and true origin in reality, from a specific source around me. Pretty wild, huh?

To further elaborate, those phantom smells, associated with delusions and schizophrenia, are the symptom of a class of psychotic and schizophrenic-like conditions which are associated almost universally with a neurological imbalance characterized by LOW GABA tone (this is why people become temporary schizophrenic-like delusionals during alcohol or benzos withdrawals); however, with high GABA tone, which is the exact opposite issue which is the one that I have, this acts as a type of condition that nootropic drugs bring on, causing enhanced, accurate perception of reality.

As for the acute abdomen, doctors have denied it and not taken the issues seriously, for the reason that no one GI doctor or surgeon has bothered to fully understand the nature of the case. They just think, "symptom, diagnosis. Symptom, diagnosis," and cannot escape or expand from this one-dimensional thinking to see the bigger picture. But my life and well-being are on the line.

Yes, I've accepted that much damage may not be reversed. I just have to live with what I have, and get the proper medical attention.

I have considered either enteroscopy or a stand-up MRI using pineapple juice as an oral contrast (very high signal on T2-weighted images due to manganese content, one can find the studies and articles on this for more info), to show if the juice flows out of the places it is supposed to be in, and into places that it isn't supposed to end up in.

Adding ghee, or any oil or fatty liquid substance, has both a positive and negative. I have considered the benefits offered by withdrawals caused by the introduction of the fats. However, for most types of lipids, proper digestion and assimilation can only occur within the digestive tract, through bile and other active liver and digestive action to conjugate the oils. For this reason, most oils may cause problems when They are allowed to escape the GI tract, in the event that they end up in extra-intestinal space, as the body may not be effective at re-uptaking and processing these oils efficiently.

However, according to my best research, the types of oils that bypass the digestive requirement for most lipjds, are MCTs, or medium-chain triglycerides (specifically C8 and C10, or capric and acrylic oils, both derived from coconut oils). These oils are of such a structure that they can diffuse through and across cells and membranes and assimilate directly into the body without the need for digestion, thus people with digestive deficiencies are able to utilize this sub-class of oils.

I have used coconut oil products and purified, refined C8/C10 oils. They also induce withdrawals.

But, eventually, symptoms inevitably return. To date, three years later, my best two theories remain: either Fasoracetam alone continues to perpetually upregulate receptor density-- and thus the perception--of increased drug effects--or, in fact, it really is true (as I believe and have accumulated more evidence for) that crystal growth is taking place, following the initial nucleations and saturation from 2016.

Doctor, your mind on this matter, I find, may be invaluable to me.

At the conclusion of your reading of this, I would like to offer to hire you, for a reasonable stipend paid regularly, to be a continued personal medical consultant to me. My friends and family are literally celebrating the fact that the first "MD" takes my case seriously. I expect that we can open many doors together.

-Jon

Hi Doctor,

I am glad to receive this good news, that you are making your way through these documents and analyzing their contents well.

You may be right about ammonia: it is well-known that one of the two primary sources of ammonia in the body comes from bacterial metabolic byproducts. This, of course, in conjunction with the symptoms and signs of continued/persistent intestinal perforations, does suggest an alarming state of things within the body.

As we know, the second source of ammonia comes in the form of metabolized amino acids that we consume. It is no coincodence, therefore, that nowadays, it is immediately after most protein-rich meals (and almost always after consuming liquid-dissolved amino acids) that I experience a huge wave of ammonia smell from my body, sweat and breath.

However, I do recall that the first waves of ammonia were felt following the olive oil flushes, implemented about a year and a half after poisoning.

The classical theory of HE correlates high degrees of ammonia with metabolic dysfunction; therefore, if my condition has been at the deepest and worst levels dueing that first year or year and a half, then why was there no perceived ammonia until this time, following the oil.immersion?

I will explain exactly why. I have not made this explanation clear in the original writings at the time, but hindsight has made things extremely clear. It is for this reason that i often consider conducting an entire re-write of this original information.

Simply put, this is what was going on:

In a normal patient, amino acids quickly break down into ammonia; following this, the ammonia is also then quickly broken down into the final urea or uric acids (cant remember which), which is the final end product found in urine.

In patients with high degrees of significant HE, that conversion of ammonia into urea is slowed and inefficient--this is what allows larger quantities of the stuff to backlog and pool for a temporary period of time.

However, even in these cases of severe HE, we take for granted that this one segment of the chain of events is the only segment affected. In other words, we take for granted that the first step--the conversion of amino acids into ammonia--is not significantly impeded, and that it is only the conversiom of ammonia that lags behind in this condition.

However, up until the first olive oil flushes, what has actually been happening, is that the VERY INTRODUCTORY PRODUCTS AND PARENT MOLECULES THEMSELVES were so completely unable to be transformed into the intermediary byproduct of ammonia due to likely-unprecedented GABAergic overwhelm, that amino acids largely remained amino acids, and became, at best, subject only to the basic intracellular metabolism of the various cells, if even that.

However, once olive oil was used en masse, for the first time, we had a liver that had the Phenibut/Fasoracetam drugs cleared away--enough to allow a sudden starting of their functions, in the way that a lawnmower suddenly starts up. Immediately after this, a massive backlog of ammonia was able to be produced, which could not yet subsequently be convertes rapidly into further waste products due to remaining systemic/toxic effects still at play.

I will need to clarify this somewhere in my writing, as this is important.

No sign of ascites could be determined, following the initial bursts. However, the opposite of ascites was onserved by me--only a reduction of abdominal volume, caused by the loss of peritoneal fluids. However, to date, no classically trained GI doc accepts this. Jowever, tjey are wrong, and perceive the truths of my case very poorly, largely for the reason that they have not taken the time to take as close of a look as you have, as well as those friends and family of mine, who now also increasingly sjow support and improved understanding.

As for the possibility of olfactory hallucinations, I am obliged to set the matter clear. In the medical community here, any doctor who reads of a case or a description of a phenomenon of olfactory hallucinations, may likely interpret this to mean a set or experience of perceived scents which DO NOT have any basis in reality.

However, if any doctor or clinician were to be allowed to think that I am perceiving scents which are merely simulated, and not based in reality, this doctor would thus be assuming the nature of my condition incorrectly.

The difference between increased perceptive capacity vs delusional perception of non-real scents, is the difference between hallucinations and increased accuity of smell.

If a medicine were created which allpws humans to rapidly grow and upregulate visual receptor density, such that the human using it could now see like a hawk, and accurately perceive and read road signs posted one kilometer away, we would not call his improved vision a hallucination, even though it is vision now beyond the range of normal human sight. This enhanced vision would be likely termed, instead, enhanced visual accuity.

The same is true for the nose senses and receptors, which is what is happening with me. Not hallucinatory--rather, a chemically-induced increase of accurate sensory accuity.

Furthermore, such a medication, in my example, does already exist--it is the combinatoon of Phenibut and Fasoracetam, which I have been poisoned with. I have already demonstrated to family around me that, when the symptoms/drugs concentrations are peaking at their highest, I am able to see very small details from very far away. Additionally, I am able to see everything in my visual periphery nearly as clearly as what is in the center of our field of vision. This allowed me to see everything placed in every window within multiple buildings in the distance, all at the same time. I could literally see everything at once. It was very overwhelming.

(To note--this capability is lowered now, after the last several months of using high dose caffeine and chocolate in my system, both being GABA inhibiting agents.)

Thank you for your continued availability, your dedication, amd your loyalty to both our work at hand, and to this website as well, which provides a wonderful service for people in my position, which complex conditions.

My mother has read up to C21 or C22. She tells me is seeing more now, through her careful reading, than she was understanding before. This is good news--but we still do not know who to turn to, or who to trust within our own local community of doctors. Ironically, in a city like New York, we find such incompetence, that we are not sure where to turn.

As a result, we feel that we may have the beat chances if we form some kind of team. We would love to have you retained as a consultant for our team, providing guidance to us wherever possibkle. This, as a result of the fact that you are literally coming into the position of being the most well-versed MD expert on my case, in this entire world!

Looking forward to hearing back from you soon.

Hi Doctor,

I am glad to receive this good news, that you are making your way through these documents and analyzing their contents well.

You may be right about ammonia: it is well-known that one of the two primary sources of ammonia in the body comes from bacterial metabolic byproducts. This, of course, in conjunction with the symptoms and signs of continued/persistent intestinal perforations, does suggest an alarming state of things within the body.

As we know, the second source of ammonia comes in the form of metabolized amino acids that we consume. It is no coincodence, therefore, that nowadays, it is immediately after most protein-rich meals (and almost always after consuming liquid-dissolved amino acids) that I experience a huge wave of ammonia smell from my body, sweat and breath.

However, I do recall that the first waves of ammonia were felt following the olive oil flushes, implemented about a year and a half after poisoning.

The classical theory of HE correlates high degrees of ammonia with metabolic dysfunction; therefore, if my condition has been at the deepest and worst levels dueing that first year or year and a half, then why was there no perceived ammonia until this time, following the oil.immersion?

I will explain exactly why. I have not made this explanation clear in the original writings at the time, but hindsight has made things extremely clear. It is for this reason that i often consider conducting an entire re-write of this original information.

Simply put, this is what was going on:

In a normal patient, amino acids quickly break down into ammonia; following this, the ammonia is also then quickly broken down into the final urea or uric acids (cant remember which), which is the final end product found in urine.

In patients with high degrees of significant HE, that conversion of ammonia into urea is slowed and inefficient--this is what allows larger quantities of the stuff to backlog and pool for a temporary period of time.

However, even in these cases of severe HE, we take for granted that this one segment of the chain of events is the only segment affected. In other words, we take for granted that the first step--the conversion of amino acids into ammonia--is not significantly impeded, and that it is only the conversiom of ammonia that lags behind in this condition.

However, up until the first olive oil flushes, what has actually been happening, is that the VERY INTRODUCTORY PRODUCTS AND PARENT MOLECULES THEMSELVES were so completely unable to be transformed into the intermediary byproduct of ammonia due to likely-unprecedented GABAergic overwhelm, that amino acids largely remained amino acids, and became, at best, subject only to the basic intracellular metabolism of the various cells, if even that.

However, once olive oil was used en masse, for the first time, we had a liver that had the Phenibut/Fasoracetam drugs cleared away--enough to allow a sudden starting of their functions, in the way that a lawnmower suddenly starts up. Immediately after this, a massive backlog of ammonia was able to be produced, which could not yet subsequently be convertes rapidly into further waste products due to remaining systemic/toxic effects still at play.

I will need to clarify this somewhere in my writing, as this is important.

No sign of ascites could be determined, following the initial bursts. However, the opposite of ascites was onserved by me--only a reduction of abdominal volume, caused by the loss of peritoneal fluids. However, to date, no classically trained GI doc accepts this. Jowever, tjey are wrong, and perceive the truths of my case very poorly, largely for the reason that they have not taken the time to take as close of a look as you have, as well as those friends and family of mine, who now also increasingly sjow support and improved understanding.

As for the possibility of olfactory hallucinations, I am obliged to set the matter clear. In the medical community here, any doctor who reads of a case or a description of a phenomenon of olfactory hallucinations, may likely interpret this to mean a set or experience of perceived scents which DO NOT have any basis in reality.

However, if any doctor or clinician were to be allowed to think that I am perceiving scents which are merely simulated, and not based in reality, this doctor would thus be assuming the nature of my condition incorrectly.

The difference between increased perceptive capacity vs delusional perception of non-real scents, is the difference between hallucinations and increased accuity of smell.

If a medicine were created which allpws humans to rapidly grow and upregulate visual receptor density, such that the human using it could now see like a hawk, and accurately perceive and read road signs posted one kilometer away, we would not call his improved vision a hallucination, even though it is vision now beyond the range of normal human sight. This enhanced vision would be likely termed, instead, enhanced visual accuity.

The same is true for the nose senses and receptors, which is what is happening with me. Not hallucinatory--rather, a chemically-induced increase of accurate sensory accuity.

Furthermore, such a medication, in my example, does already exist--it is the combinatoon of Phenibut and Fasoracetam, which I have been poisoned with. I have already demonstrated to family around me that, when the symptoms/drugs concentrations are peaking at their highest, I am able to see very small details from very far away. Additionally, I am able to see everything in my visual periphery nearly as clearly as what is in the center of our field of vision. This allowed me to see everything placed in every window within multiple buildings in the distance, all at the same time. I could literally see everything at once. It was very overwhelming.

(To note--this capability is lowered now, after the last several months of using high dose caffeine and chocolate in my system, both being GABA inhibiting agents.)

Thank you for your continued availability, your dedication, amd your loyalty to both our work at hand, and to this website as well, which provides a wonderful service for people in my position, which complex conditions.

My mother has read up to C21 or C22. She tells me is seeing more now, through her careful reading, than she was understanding before. This is good news--but we still do not know who to turn to, or who to trust within our own local community of doctors. Ironically, in a city like New York, we find such incompetence, that we are not sure where to turn.

As a result, we feel that we may have the beat chances if we form some kind of team. We would love to have you retained as a consultant for our team, providing guidance to us wherever possibkle. This, as a result of the fact that you are literally coming into the position of being the most well-versed MD expert on my case, in this entire world!

Looking forward to hearing back from you soon.

Hi Doctor,

I am glad to hear you have read so far into the set. The C22 sub-set, the following document mini-set, is the same conversation and presentation on ammonia and HE diagnosis, that you have already read originally.

I am curious, now on a second read-thru after having read more about my case, whether a doctor in your position may see slightly more connections and relevance about the HE theory and candidacy for diagnosis, that may not have been picked up the first time.

After that, C23 is a short symptoms list, which you have also read, and then C24 is the last fully-prepared update, covering most of the updates for this year.

I am currently working on C25 (slowly, I must admit)--although only one of two days of active, focused work remain before I am finished with C25 as well.

As for the source of ammonia, it is true that there may be a uniqu mixture of ammonia-generating sources, including catabolism from tissues. It is indeed true that I have lost muscle mass since being poisoned with Phenibut and Fasoracetam, given that they seem to inhibit the maintenance of large amounts of strength and muscle mass that I used to possess.

However, it may not necessary be the source of the ammonia that is most critical in the diagnosis of hepatic or metabolic dysfunction, but rather, the evaluation of how effective the body is at dealing with any potential surplus.

You may recall from the C22 set of docs that in the best studies on ammnia (one of which I cited from, and a second that comes to mind which I may not have discussed as deeply in the material, unfortunately), an oral glutamine challenge was shown to produce significant and excessive "back-logs" of pooled ammonia in the body almost exclusively in those patients with apparent dysfunction--and almost never in the control patient that were healthy. It is not as much the absolute ammonia levels that are relevant, studies find, as are the critical nature of the change and increase in ammonia in response to precursors that would generate much ammonia. Regardless, we can recall that when my own ammonia was tested, it reached as high as 60 without an OGC (average is 30-ish w/o an OGC for healthy controls), and as high as 105 (+45 higher net increase for me) with an OGC. (average is 33-ish--an increase of only +3--w/ an OGC for healthy controls).

Healthy controls only increased their ammonia by +3, on average, following an OGC, but nhealthy contorls found theirs to shoot up to +44. That is a remarkable difference--and my net increase happens to match almost exactly the increase amounts found in non-healthy population. This is, of course, not a coincidence.

. . .

You state that there are often additional symptoms that are present in those patients who have hyperammonia. What are they?

In brief conversations with other doctors (not just the one you have seen who is from this website), I have had it suggested to be that "I could not possibly have high ammonia and/or H.E., because if I did, I wouldn't be able to sit and write such lengthy documents.

However, I wish to make something very clear: There is ne profound difference between myself and most other patients with reduced functionality of the drug-metabolizing microsomes of the liver.

This critical difference, is this:

Most sufferers of excessive GABA-ergic tone are suffering from an excessive stimuation of GABA-A receptors in particular. It is due to this that, in addition to having the CNS activity lowered to such an extent that the microsomes are disabled, the levels of consciousness are also reduced. ***THIS IS BECAUSE GABA-A AGONISTS, SUCH AS ALCOHOL, BENZODIAZEPINES, BARBITURATES, ETC, ALL CAUSE EXCESSIVE DROWSINESS, SEDATION, AND LOSS OF CONSCIOUSNESS, MEMORY AND FUNCTIONALITY AT HIGH LEVELS.***

This is why it is assumed that cognitive tests may be a supposedly effective method of diagnosing acute instances of hyperammonia or HE, although multiple experts commenting on this, mentinoed in C22c or C22d, admit that it is not the best approach, and that there are shortcomings and limitations with such a diagnostic route.

However, my situation is different: Phnibut and Fasoracetam are increasers of GABA-B tone specifically. This is why chemicals which include affinity for GABA-B, such as Phenibut, Fasoracetam, as well as orally-dosed form of the actual neurotransmitter GABA itself (!!), which can be bought in stores and taken as a supplement, are NOT associated with sedation to nearly the same degree.

With a case of alcohol, benzo or barbiturate poisoning, by the time you get to high-enough concentrations to see a lowered respiratory rate down to 6 - 8 breaths per minute or less, the level of consciousness of the person is so low that he is likely unconscious and cannot function or be alert.

However, because I am poisoned with excessive GABA-B tone specifically, I retain all the signs of lowered CNS activity (bradypnea, apparent inhibition of drug-metabolizing microsomes, apparent inhibition of full kidney function, etc)--but al of this happens while I remain completely, fully conscious and functional; this masks the seriousness of the underlying condition, and thus is a severe impediment and blockage to the proper diagnosis of my condition.

. . .

As for my increased perception in general, which we have mentioned, this is an apparent function of the drug poisoning. However, it is important for to once again note, that as far as i can tell (with very acute and accurate internal sensory, if I may say so myself), there is no evidence of there ever being created any kind of "simulated" or false perception of any sensation, neural or otherwise, within my system or through any of the senses.

Specifically, with regards to the sensations felt of cold water pooling up at the pelvic floor (and now, most recently, even beyond this, as you will find mentioned in C24), it has been suggested to me bymy own doctor that this could be nothing more thatn a "reference sensation," or some false generation of perceptive signals caused by the nerves.

This is his theory, because he cannot possibly believe that I have been walking around for over a year--while still alive, and not in pain and with no inflammation (although I state clearly that signs of infection have been present and have already caused apparent damage)--with an acute-on-chronic abdominal perforation. However, I can virtually guarantee that it is completely, literally true. Of all the theories and assertions I make about my own case, including Phenibut theory, growth theory and toxicity, and H.E--I am literally more sure of the fact that there has been long-standing intestinal perforations than of anything else. i am more sure of this, than I am of my own name--and I am someone who is well-known to be very logical, careful and exact with my words and conclusions.

You have mentioned that intestinal tissue normally heals and seals within a few days. This is something I find to be true, and definitely do agree with; I have found it to be the case that with most of the acute tears experienced, within a few days to a few weeks the live intestinal tissue does a remarkable job of constantly trying to re-seal completely.

However, the situation is complicated for this reason:

The active agent causing tears, is the unmetabolized cocaine saturating the tissues. Because the liver does not metabolize these, and the body is thus ineffective at excreting this dangerous agent, it continues to re-cycle and re-circulate between tissues, circulation, and deep fatty storage.

As a result...

***NEARLY EVERY TIME I ENGAGE IN A HIGHLY LIPOLYTIC ACTIVITY, THE COCAINE IS RE-RELEASED EN MASSE***--after which point, a new tear is found within HOURS after lipolysis after drinking a cold beverage, and discovering a new opening or tear that has formed. Because the intestinal tissue cannot escape the presence of the cocaine within the physical system, the cocaine continually causes both re-occuring damage, but more importantly, it also blocks healing by the very same property of tissue saturation.

This is extremely important, and is not something I mention to a great extent in my writing. What do I mean by inhibited healing caused by drug saturation of the tissues?

Imagine this: Have you ever seen drug addicts, such as heroin addicts or other similar types of drugs, with sores and wounds on their body? How often do you find that, in these cases, these sores and wounds tends to be very delayed and slow in healing?

The reason for this is because the drugs are dosed so often, that the drugs physically and literally saturate the tissues of th flesh, nearly uniformly, all thoughout the body. With my super-sense of smell, I have been able to smell and detect this phenomenon, while also pysically touching and examining a couple of people in that situation, where i found that as a result of their drug saturation, their skin all thoughout their body smells like dried paper with the smell of the drug.

Imagine two wet paper towels--one absorbs only distilled water, then the water is allowed to evaporate and dry; the second paper towel is soaked with water that has a very large amount of salt dissolved in it! Then, this second paper towel is also made to dry.

When you touch the first paper towel, you would find that by touch, its softness has been fully retained. However, in the case of the second paper towel, you would find it is dry, rigid, and crispy to the touch; moreover, you would know that this quality isnt the result of a hypothetical or theoretical dehydration that is systemic--rather, you know know that it is literally the saturation of the dried, supersaturated solute itself, which you touch and make contact with on the surface of the paper towel, that causes the rigidification. It is this same drug-soaked tissue rigidification that petrifies the tissue of the drug user, and blocks healing and nourishment of the wounds--and it is the same "cocaine-crispiness" of the actual intestinal tissues themselves at the perforation sites, that preserves the tears and leaves them open for an extended period of time.

Moreover, it was found that after squirting large amounts of coconut oil into my intestinal tract, tears that were opened for weeks and months were found, within the mere hours and days that followed, to have re-sealed. This was because the oils allowed the pulling and tranferance of saturated drug matter out from those localized tissues, allowing immedaite resensitization and reawareness in the nervous system, increased blood flow and hydration, prompting healing immedaitely thereafter.

However, even this was temporary, as more fat-stored drugs being released caused subsequent eventual tears.

I have had one MRE done, which was performed without IV contrast (which I declined for obvious reasons). Pineapple juice was used for oral contrast. However, the report says that results are unclear, and thus implying that nothing can be ruled out.

A deeper explanation is presented in my writing, where i discuss how collapsed small bowels, as found in my case, are actually indicative and highly suggestive of a tear.

. . .

Fungal infections can be worrisome. However, besides the DNA damage that they are known to cause, their only other worst symptoms and threat is mediated by damage caused by excessive, uncontrolled inflammation induced systemically by the presence of the spreading infections and the CNS's automatic response to them. However, since I have drastically reduced inflammatory capacity, it is almost like a kind of ironic blessing: in this state, the infections live in my system without causing the types of reactions that are known to be often fatal in regular people. So I just live on with my life.

Furthermore, the use of Fluconazole does concern me: besides issues of dryness, I am wary of its known, studied effects of LIVER/MICROSOMAL INHIBITION of liver metabolic pathways--this is the opposite of what I need--to be poisoned with something that further inhibits liver function.

I agree with you that the treatment of the fungal infection may be lower priority at this time. I would wait until all drugs are pulled out of my system via hemoperfusion/MARS, assisted by sauna sweating, fasting and acidification/conjugation/solubility-increasing agents.

. . .

My mom just finished reading up thru C24. She and I wouuld like to start a project where we conduct a type of private "mock trial" to determine the truths of my case as best we can. We may then take the conclusions we find, and conduct a type of interview series that we can post onto YouTube, so that others can easily learn about my case and also realize its undeniable truths.

We look forward to hearing more from you. You are a blessing to us!!


. . .

For entertainment purposes, I ask that you watch two wonderful movies whenever you have some free time. They are (1) Limitless (2011) and XXXXXXX (2014). They are both exaggerations and parodies of the kind of neural an cognitive enhancements that I have experienced--and it is no joke when I tell you, that at the very highest drug levels of these, the kinds of enhancements depicted in the movie Limitless would only be a moderate over-exaggeration aat best, compared to the real, tangible and astonishing enhancements I would experience as a result of the combination of these drugs.

Hi Doctor,

I am glad to hear you have read so far into the set. The C22 sub-set, the following document mini-set, is the same conversation and presentation on ammonia and HE diagnosis, that you have already read originally.

I am curious, now on a second read-thru after having read more about my case, whether a doctor in your position may see slightly more connections and relevance about the HE theory and candidacy for diagnosis, that may not have been picked up the first time.

After that, C23 is a short symptoms list, which you have also read, and then C24 is the last fully-prepared update, covering most of the updates for this year.

I am currently working on C25 (slowly, I must admit)--although only one of two days of active, focused work remain before I am finished with C25 as well.

As for the source of ammonia, it is true that there may be a uniqu mixture of ammonia-generating sources, including catabolism from tissues. It is indeed true that I have lost muscle mass since being poisoned with Phenibut and Fasoracetam, given that they seem to inhibit the maintenance of large amounts of strength and muscle mass that I used to possess.

However, it may not necessary be the source of the ammonia that is most critical in the diagnosis of hepatic or metabolic dysfunction, but rather, the evaluation of how effective the body is at dealing with any potential surplus.

You may recall from the C22 set of docs that in the best studies on ammnia (one of which I cited from, and a second that comes to mind which I may not have discussed as deeply in the material, unfortunately), an oral glutamine challenge was shown to produce significant and excessive "back-logs" of pooled ammonia in the body almost exclusively in those patients with apparent dysfunction--and almost never in the control patient that were healthy. It is not as much the absolute ammonia levels that are relevant, studies find, as are the critical nature of the change and increase in ammonia in response to precursors that would generate much ammonia. Regardless, we can recall that when my own ammonia was tested, it reached as high as 60 without an OGC (average is 30-ish w/o an OGC for healthy controls), and as high as 105 (+45 higher net increase for me) with an OGC. (average is 33-ish--an increase of only +3--w/ an OGC for healthy controls).

Healthy controls only increased their ammonia by +3, on average, following an OGC, but nhealthy contorls found theirs to shoot up to +44. That is a remarkable difference--and my net increase happens to match almost exactly the increase amounts found in non-healthy population. This is, of course, not a coincidence.

. . .

You state that there are often additional symptoms that are present in those patients who have hyperammonia. What are they?

In brief conversations with other doctors (not just the one you have seen who is from this website), I have had it suggested to be that "I could not possibly have high ammonia and/or H.E., because if I did, I wouldn't be able to sit and write such lengthy documents.

However, I wish to make something very clear: There is ne profound difference between myself and most other patients with reduced functionality of the drug-metabolizing microsomes of the liver.

This critical difference, is this:

Most sufferers of excessive GABA-ergic tone are suffering from an excessive stimuation of GABA-A receptors in particular. It is due to this that, in addition to having the CNS activity lowered to such an extent that the microsomes are disabled, the levels of consciousness are also reduced. ***THIS IS BECAUSE GABA-A AGONISTS, SUCH AS ALCOHOL, BENZODIAZEPINES, BARBITURATES, ETC, ALL CAUSE EXCESSIVE DROWSINESS, SEDATION, AND LOSS OF CONSCIOUSNESS, MEMORY AND FUNCTIONALITY AT HIGH LEVELS.***

This is why it is assumed that cognitive tests may be a supposedly effective method of diagnosing acute instances of hyperammonia or HE, although multiple experts commenting on this, mentinoed in C22c or C22d, admit that it is not the best approach, and that there are shortcomings and limitations with such a diagnostic route.

However, my situation is different: Phnibut and Fasoracetam are increasers of GABA-B tone specifically. This is why chemicals which include affinity for GABA-B, such as Phenibut, Fasoracetam, as well as orally-dosed form of the actual neurotransmitter GABA itself (!!), which can be bought in stores and taken as a supplement, are NOT associated with sedation to nearly the same degree.

With a case of alcohol, benzo or barbiturate poisoning, by the time you get to high-enough concentrations to see a lowered respiratory rate down to 6 - 8 breaths per minute or less, the level of consciousness of the person is so low that he is likely unconscious and cannot function or be alert.

However, because I am poisoned with excessive GABA-B tone specifically, I retain all the signs of lowered CNS activity (bradypnea, apparent inhibition of drug-metabolizing microsomes, apparent inhibition of full kidney function, etc)--but al of this happens while I remain completely, fully conscious and functional; this masks the seriousness of the underlying condition, and thus is a severe impediment and blockage to the proper diagnosis of my condition.

. . .

As for my increased perception in general, which we have mentioned, this is an apparent function of the drug poisoning. However, it is important for to once again note, that as far as i can tell (with very acute and accurate internal sensory, if I may say so myself), there is no evidence of there ever being created any kind of "simulated" or false perception of any sensation, neural or otherwise, within my system or through any of the senses.

Specifically, with regards to the sensations felt of cold water pooling up at the pelvic floor (and now, most recently, even beyond this, as you will find mentioned in C24), it has been suggested to me bymy own doctor that this could be nothing more thatn a "reference sensation," or some false generation of perceptive signals caused by the nerves.

This is his theory, because he cannot possibly believe that I have been walking around for over a year--while still alive, and not in pain and with no inflammation (although I state clearly that signs of infection have been present and have already caused apparent damage)--with an acute-on-chronic abdominal perforation. However, I can virtually guarantee that it is completely, literally true. Of all the theories and assertions I make about my own case, including Phenibut theory, growth theory and toxicity, and H.E--I am literally more sure of the fact that there has been long-standing intestinal perforations than of anything else. i am more sure of this, than I am of my own name--and I am someone who is well-known to be very logical, careful and exact with my words and conclusions.

You have mentioned that intestinal tissue normally heals and seals within a few days. This is something I find to be true, and definitely do agree with; I have found it to be the case that with most of the acute tears experienced, within a few days to a few weeks the live intestinal tissue does a remarkable job of constantly trying to re-seal completely.

However, the situation is complicated for this reason:

The active agent causing tears, is the unmetabolized cocaine saturating the tissues. Because the liver does not metabolize these, and the body is thus ineffective at excreting this dangerous agent, it continues to re-cycle and re-circulate between tissues, circulation, and deep fatty storage.

As a result...

***NEARLY EVERY TIME I ENGAGE IN A HIGHLY LIPOLYTIC ACTIVITY, THE COCAINE IS RE-RELEASED EN MASSE***--after which point, a new tear is found within HOURS after lipolysis after drinking a cold beverage, and discovering a new opening or tear that has formed. Because the intestinal tissue cannot escape the presence of the cocaine within the physical system, the cocaine continually causes both re-occuring damage, but more importantly, it also blocks healing by the very same property of tissue saturation.

This is extremely important, and is not something I mention to a great extent in my writing. What do I mean by inhibited healing caused by drug saturation of the tissues?

Imagine this: Have you ever seen drug addicts, such as heroin addicts or other similar types of drugs, with sores and wounds on their body? How often do you find that, in these cases, these sores and wounds tends to be very delayed and slow in healing?

The reason for this is because the drugs are dosed so often, that the drugs physically and literally saturate the tissues of th flesh, nearly uniformly, all thoughout the body. With my super-sense of smell, I have been able to smell and detect this phenomenon, while also pysically touching and examining a couple of people in that situation, where i found that as a result of their drug saturation, their skin all thoughout their body smells like dried paper with the smell of the drug.

Imagine two wet paper towels--one absorbs only distilled water, then the water is allowed to evaporate and dry; the second paper towel is soaked with water that has a very large amount of salt dissolved in it! Then, this second paper towel is also made to dry.

When you touch the first paper towel, you would find that by touch, its softness has been fully retained. However, in the case of the second paper towel, you would find it is dry, rigid, and crispy to the touch; moreover, you would know that this quality isnt the result of a hypothetical or theoretical dehydration that is systemic--rather, you know know that it is literally the saturation of the dried, supersaturated solute itself, which you touch and make contact with on the surface of the paper towel, that causes the rigidification. It is this same drug-soaked tissue rigidification that petrifies the tissue of the drug user, and blocks healing and nourishment of the wounds--and it is the same "cocaine-crispiness" of the actual intestinal tissues themselves at the perforation sites, that preserves the tears and leaves them open for an extended period of time.

Moreover, it was found that after squirting large amounts of coconut oil into my intestinal tract, tears that were opened for weeks and months were found, within the mere hours and days that followed, to have re-sealed. This was because the oils allowed the pulling and tranferance of saturated drug matter out from those localized tissues, allowing immedaite resensitization and reawareness in the nervous system, increased blood flow and hydration, prompting healing immedaitely thereafter.

However, even this was temporary, as more fat-stored drugs being released caused subsequent eventual tears.

I have had one MRE done, which was performed without IV contrast (which I declined for obvious reasons). Pineapple juice was used for oral contrast. However, the report says that results are unclear, and thus implying that nothing can be ruled out.

A deeper explanation is presented in my writing, where i discuss how collapsed small bowels, as found in my case, are actually indicative and highly suggestive of a tear.

. . .

Fungal infections can be worrisome. However, besides the DNA damage that they are known to cause, their only other worst symptoms and threat is mediated by damage caused by excessive, uncontrolled inflammation induced systemically by the presence of the spreading infections and the CNS's automatic response to them. However, since I have drastically reduced inflammatory capacity, it is almost like a kind of ironic blessing: in this state, the infections live in my system without causing the types of reactions that are known to be often fatal in regular people. So I just live on with my life.

Furthermore, the use of Fluconazole does concern me: besides issues of dryness, I am wary of its known, studied effects of LIVER/MICROSOMAL INHIBITION of liver metabolic pathways--this is the opposite of what I need--to be poisoned with something that further inhibits liver function.

I agree with you that the treatment of the fungal infection may be lower priority at this time. I would wait until all drugs are pulled out of my system via hemoperfusion/MARS, assisted by sauna sweating, fasting and acidification/conjugation/solubility-increasing agents.

. . .

My mom just finished reading up thru C24. She and I wouuld like to start a project where we conduct a type of private "mock trial" to determine the truths of my case as best we can. We may then take the conclusions we find, and conduct a type of interview series that we can post onto YouTube, so that others can easily learn about my case and also realize its undeniable truths.

We look forward to hearing more from you. You are a blessing to us!!


. . .

For entertainment purposes, I ask that you watch two wonderful movies whenever you have some free time. They are (1) Limitless (2011) and XXXXXXX (2014). They are both exaggerations and parodies of the kind of neural an cognitive enhancements that I have experienced--and it is no joke when I tell you, that at the very highest drug levels of these, the kinds of enhancements depicted in the movie Limitless would only be a moderate over-exaggeration aat best, compared to the real, tangible and astonishing enhancements I would experience as a result of the combination of these drugs.