Question: Without going into my detailed history right now, I'd like to obtain a complete and comprehensive list of all the possible causes for "bitemporal slowing" as indicated on a combined 23-minute EC and EO Electroencephalogram. The detail of the Neurologist's report include the following. IMPRESSION: This is an abnormal, awake and drowsy EEG due to bitemporal slowing. CLINICAL INTERPRETATION: The abnormalities are consistent with focal cortical neuronal dysfunction of the bitemporal regions which could indicate mild hypoxic/ischaemia, mass legions, metabolic derangements, medication effects, or some other cause. There were no eletrographic seizures or epileptiform activity.

A meaningful, relatively recent diagnosis has been "parasympathetic excess" (formerly known as paradoxical parasympathetic syndrome). But there is no known treatment path to address this unique autonomic dysfunctioning.

Regarding the possible causes mentioned in the report, I have never had a hypoxic episode, my two brain MRIs (2002, 2008) ruled out tumors or legions, I doubt it's metabolic derangement since I'm 6 feet tall and weight 175, and I didn't start taking dextroamphetamine UNTIL I became debilitated; and I've gone off of it many times in the past 19 years with no change, therefore I don't believe it's medication related.

So what I really need, is a COMPLETE list of every possible thing that is known to cause bitemporal slowing, not just the "most common" causes.

Something that is indisputably connected to the underlying cause of my overall dysfunction (and by extension, this clinical finding of bitemporal slowing) is the chronic instability of my atlanto-occipital joint (since 1987). The more mis-aligned, mis-rotated, mis-positioned I am here, the less functional I am overall. (C1/C2 positions visualized with x-rays.)

Dr. Panwar,

Thank you for your reply.

You listed 8 possible causes for bitemporal slowing. I believe I understand how numbers 1 through 6 might lead to this condition, but not numbers 7 and 8. Could you please explain what the dysfunctional mechanism would be in the case of an Autoimmune System cause, and in the case of a Trauma-induced cause?

Thank you very much.


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