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Why Are Symptoms Of Postprandial Anxiety, Tachycardia, Hypertension Always Investigated Through The Lens Of Hypoglycemia As In An Adrenergic Response?
I am curious why symptoms of postprandial anxiety, tachycardia, hypertension, etc are always investigated through the lens of hypoglycemia as in an adrenergic response. Wouldn't cholecystokinin, specifically CCK2 or CCB, be a better differential for these symptoms? Patients generally get labeled with Postprandial Syndrome and most claim that it doesn't exist, or is strictly an anxiety disorder. It seems as though the CNS role of CCK is overlooked in these investigations.
Wed, 13 Nov 2013
Dear sir
Not like every time we should diagnose hypoglycemia as common.
But in diabetes if the patient is taking insulin and oral hypoglycemic drugs they are easily get in to low sugars rather than other syndromes.
If low Sugars are excluded then we then think of next problems
Thanks
Not like every time we should diagnose hypoglycemia as common.
But in diabetes if the patient is taking insulin and oral hypoglycemic drugs they are easily get in to low sugars rather than other syndromes.
If low Sugars are excluded then we then think of next problems
Thanks
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